The alternate pathway of complement activation
is only seen during a secondary response
requires interactions with antibodies.
inhibits C3 and MAC formation.
is faster than the classical pathway.
is activated when factors P, B, and D in the blood interact with sugars on bacteria membranes.
is activated when factors P, B, and D in the blood interact with sugars on bacteria membranes.
The information you provided is partly correct. The alternate pathway of complement activation is indeed faster than the classical pathway. However, it is not only seen during a secondary response; both the classical and alternate pathways can be activated during primary and secondary responses.
Furthermore, the alternate pathway does not require interactions with antibodies. Unlike the classical pathway, which is antibody-dependent, the alternate pathway can be activated independently of antibodies.
Regarding the inhibition of C3 and MAC (membrane attack complex) formation, the alternate pathway does not directly inhibit C3 and MAC formation. Instead, it promotes the activation and formation of C3b, which then participates in the assembly of the MAC.
Lastly, your statement about factors P, B, and D interacting with sugars on bacterial membranes is correct. These factors, along with factor D, interact with certain sugars or carbohydrates on the surface of bacteria, leading to the activation of the alternate pathway.